Hehe kar bodite poskusni zajci. Bomo videli na stara leta kako se vam bo tole "mastenje" obrestovalo. Pa lepo prosim, da ne govorite o OH kot skupini, ki je povezana samo z belo moko, sladkorjem in nasplošno procesirano hrano. Kot tudi maščobe niso vse enako blagodejne za telo. In btw, ko ješ neko živilo ne dobiš samo tistih OH-jev (recimo sadje) ampak še vse drugo kar sadež (ali drugo živilo) ponuja.
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Hja, veš kako je...če pa jem tako kot "se priporoča" pa me čaka kaj...pogledam okoli, starostnike in tiste ki sem jih spremljal v zadnjih urah...polno kognitivnih obolenj, CV je v drami, rak, etc etc... torej če jem tako kot se priporoča me ne čaka kaj preveč blesteča prihodnost in zelo verjetno smrt v mukah.
Edino če sem zadel na genetskem lotu, ampak s tem ne bi tvegal
Edino če sem zadel na genetskem lotu, ampak s tem ne bi tvegal
No kaj pa se priporoča?
lej tole http://www.nutris.org/prehrana/abc-prehrane/splosno/94-smernice-zdrave-prehrane.html
lej tole http://www.nutris.org/prehrana/abc-prehrane/splosno/94-smernice-zdrave-prehrane.html
Jaz bi rekel, da je švedska bolj na pravi poti: http://healthimpactnews.com/20...-fat-nutrition/
Potem na japonskem se tudi nekaj premaknilo letos, celo odvrgli so tezo o "hudičevem holesterolu" in ga tako več ne kontrolirajo, kot se pri nas. torej nimajo več max vrednosti holesterola, po kateri potem zaženejo paniko in dajo človeka na tablete, ampak gledajo druge parametre.
Splošno rečeno pa je to bolj LCHF stil, kot pa OH stil.
Potem na japonskem se tudi nekaj premaknilo letos, celo odvrgli so tezo o "hudičevem holesterolu" in ga tako več ne kontrolirajo, kot se pri nas. torej nimajo več max vrednosti holesterola, po kateri potem zaženejo paniko in dajo človeka na tablete, ampak gledajo druge parametre.
Splošno rečeno pa je to bolj LCHF stil, kot pa OH stil.
Hah ma ne ga srat http://www.independent.co.uk/l...en-7836468.html
Aja sej res holesterol pri srčno žilnih obolenjih nima veze. Pa prosim pusti razne blogspote, health-blabla domene pri miru. Limaj mi recimo iz pubmed-a.
Aja sej res holesterol pri srčno žilnih obolenjih nima veze. Pa prosim pusti razne blogspote, health-blabla domene pri miru. Limaj mi recimo iz pubmed-a.
Nazadnje urejeno:
Nisem govoril o blogspotih, ampak o japonskih znanstvenikih. Ena študija je tudi povezala višino holesterola z dolgoživostjo, torej več ga imaš, dlje živiš:
http://www.drbriffa.com/2012/0...rol-hypothesis/
Japanese Research Exposes Statin Scam: People with High Cholesterol Live Longer
http://healthimpactnews.com/20...ol-live-longer/
In epilog:
Japanese Government Drops Cholesterol Limit in Health Guidance:
http://blogs.wsj.com/japanreal...ealth-guidance/
Moraš tudi vedet, da je posel s statini zelo dobičkonosen, majhna kost za vse iskalce zarot.
http://www.drbriffa.com/2012/0...rol-hypothesis/
Japanese Research Exposes Statin Scam: People with High Cholesterol Live Longer
http://healthimpactnews.com/20...ol-live-longer/
In epilog:
Japanese Government Drops Cholesterol Limit in Health Guidance:
http://blogs.wsj.com/japanreal...ealth-guidance/
Moraš tudi vedet, da je posel s statini zelo dobičkonosen, majhna kost za vse iskalce zarot.
Če sam nič ne narediš, da si izboljšaš zdravje in vrednosti holesterola pa jej tablete. Vsaka ti čast, da svoje zdravje zaupaš eni?! študiji japanskih znanstvenikov v okolju kjer je itak kultura in prehranjevanje drugačno od našega, medtem, ko obstaja malo morje dokazov proti. Samo ne, vsi so podkupljeni. Maš cojones ampak tudi take rabimo.
Ja, fino ane. Ketonska dieta naj bi zniževala vrednost holesterola http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2716748/
še posebej LDL, japanska študija pa trdi da višje vrednosti prispevajo k manjši umrljivosti. Se boš moral še odločit katera je boljša zate.
še posebej LDL, japanska študija pa trdi da višje vrednosti prispevajo k manjši umrljivosti. Se boš moral še odločit katera je boljša zate.
Bi bilo potrebno detajlno pogledat to študijo, ker mnoge kažejo drugače:
In a study from 2009 by Brinkworth et al., researchers recruited 118 obese men and women and separated them into two groups for a year-long intervention study. The first group consumed a very-low-carbohydrate, high-saturated diet with 4%, 35%, and 61% energy from carbohydrates, proteins, and fats respectively.
This equates to about 20 grams of carbohydrates on a 2000 kcal diet per day – a more stringent application of carbohydrate restriction use than the meta-analysis above. The second group consumed primarily a low-fat diet with 46%, 24%, and 30% energy from respectively from carbohydrate, protein, and fat per day. Both diets contained an equal number of calories.
About 59% of participants completed the 12-month trial: 33 in the low-carb group and 36 in the low-fat group. Both groups lost similar amount of body weight and body fat. The subjects who participated in the low-fat intervention experienced an average increase in HDL cholesterol of 0.07 mmol/L from 1.36 mmol/L to 1.43 mmol/L. [12] This represents a 4.9% increase.
In contrast, the subjects who participated in the high-fat intervention experienced an average increase in HDL cholesterol of 0.30 mmol/L from 1.45 mmol/L to 1.75 mmol/L. [12] This represents a 20.6% increase – almost four times as much as that found in the low-fat diet.
In a study from 2009 by Brinkworth et al., researchers recruited 118 obese men and women and separated them into two groups for a year-long intervention study. The first group consumed a very-low-carbohydrate, high-saturated diet with 4%, 35%, and 61% energy from carbohydrates, proteins, and fats respectively.
This equates to about 20 grams of carbohydrates on a 2000 kcal diet per day – a more stringent application of carbohydrate restriction use than the meta-analysis above. The second group consumed primarily a low-fat diet with 46%, 24%, and 30% energy from respectively from carbohydrate, protein, and fat per day. Both diets contained an equal number of calories.
About 59% of participants completed the 12-month trial: 33 in the low-carb group and 36 in the low-fat group. Both groups lost similar amount of body weight and body fat. The subjects who participated in the low-fat intervention experienced an average increase in HDL cholesterol of 0.07 mmol/L from 1.36 mmol/L to 1.43 mmol/L. [12] This represents a 4.9% increase.
In contrast, the subjects who participated in the high-fat intervention experienced an average increase in HDL cholesterol of 0.30 mmol/L from 1.45 mmol/L to 1.75 mmol/L. [12] This represents a 20.6% increase – almost four times as much as that found in the low-fat diet.
Hudič je v detajlih, to sem že pisal, da LDL vpliva na imunski sistem in da sploh ni tako slab, kot ga prikazuje "naše zdravstvo", LDL - slab, HDL - dober. Samo najmanjši LDL delci so tisti, ki mašijo žile.
To je ena primerja pred in po ketogenski dieti:
Lepo se vidi, da je največji efekt na znižanju najmanjših delcev, "dobre" zadeve pa so se malce povišale.
To je ena primerja pred in po ketogenski dieti:
Lepo se vidi, da je največji efekt na znižanju najmanjših delcev, "dobre" zadeve pa so se malce povišale.
http://www2.lbl.gov/Science-Articles/Archive/extremediet-patternb.html
Poglej, js sm vedno zagovarjal zmerno uravnoteženo prehrano. ti pa se vrtiš v extremih, torej izločaš kar cele skupine hranil.
The interaction between diet and genes and how this interaction affects the two LDL subclass patterns may help to explain why there can be so much variability in the effects of low-fat diets on the risk of heart disease. For patients who started out with the pattern B cholesterol profile, the study by Krauss and his research group showed that an extreme low-fat/high-carbohydrate diet can reduce the number of circulating small LDL particles which in turn reduces the risk of heart disease. LDL particles carry cholesterol through the blood stream and deposit it along arterial walls, creating a waxy buildup that can eventually restrict the flow of blood to the heart.
For patients who started out with the pattern A cholesterol profile, however, Krauss and his research group found that an extreme low- fat/high-carbohydrate diet worked to reduce the cholesterol content of the LDL particles circulating in the blood. This depletion in the composition of the LDL particles resulted in a downsizing that in turn led to a conversion from the pattern A to the pattern B profile.
Ni vse za vsakega, še posebej pa ne ekstremi katere zagovarjaš ti.
Torej še enkrat http://mainheartclinic.com/2011/05/cholesterol-particle-size/
Even though LDL cholesterol particle size is mainly genetically inherited, individuals who have small LDL particles (pattern B) can increase their particle size through diet, exercise, and medications.
Diets that are low in saturated fat and cholesterol, regular aerobic exercise, and loss of excess body fat have been determined to decrease the number of small LDL particles and increase the number of large LDL particles in the blood. In other words, lifestyle modifications can change pattern B to pattern A.
Poglej, js sm vedno zagovarjal zmerno uravnoteženo prehrano. ti pa se vrtiš v extremih, torej izločaš kar cele skupine hranil.
The interaction between diet and genes and how this interaction affects the two LDL subclass patterns may help to explain why there can be so much variability in the effects of low-fat diets on the risk of heart disease. For patients who started out with the pattern B cholesterol profile, the study by Krauss and his research group showed that an extreme low-fat/high-carbohydrate diet can reduce the number of circulating small LDL particles which in turn reduces the risk of heart disease. LDL particles carry cholesterol through the blood stream and deposit it along arterial walls, creating a waxy buildup that can eventually restrict the flow of blood to the heart.
For patients who started out with the pattern A cholesterol profile, however, Krauss and his research group found that an extreme low- fat/high-carbohydrate diet worked to reduce the cholesterol content of the LDL particles circulating in the blood. This depletion in the composition of the LDL particles resulted in a downsizing that in turn led to a conversion from the pattern A to the pattern B profile.
Ni vse za vsakega, še posebej pa ne ekstremi katere zagovarjaš ti.
Torej še enkrat http://mainheartclinic.com/2011/05/cholesterol-particle-size/
Even though LDL cholesterol particle size is mainly genetically inherited, individuals who have small LDL particles (pattern B) can increase their particle size through diet, exercise, and medications.
Diets that are low in saturated fat and cholesterol, regular aerobic exercise, and loss of excess body fat have been determined to decrease the number of small LDL particles and increase the number of large LDL particles in the blood. In other words, lifestyle modifications can change pattern B to pattern A.
Sigurno je človek bolje fizično in genetsko prilagojen na hrano, ki jo je jedel 2 miljona let, kot pa na hrano iz zadnjih par tisoč let. Res je pa da brez tega(poljedelstva), ne bi bilo civilizacije kot jo poznamo...prav tako pa ne raznoraznih degenerativnih bolezni.
Imaš kakšne raziskave ali podatke, ki bi podkrepili to izjavo, ali pa vsaj razloži, zakaj bi tako mislil?
Drugače pa, če recimo gledamo rakave celice/tumorje, le-ti potrebujejo glukozo za preživetje/množenje, ker imajo defektne mitohondrije in ne morejo uporabiti BHB (ketone) za vir energije. In obstaja več študij, ki dokazuje, da ketogenska dieta zavira/preprečuje razvoj rakastih celic/tumorjev, oz v nekaterih primerih celo "ozdravi" človeka. Poleg tega pa deluje blagodejno za srčno mišico in za možgane, možgani imajo dejansko rajši BHB kot vir energije, glede na glukozo.
Torej dva naša najbolj pomembna organa preferirata ketone, najbolj zahrbtne moderne bolezni (rak, alzheimer, ipd) se lahko s tem preprečijo in še sila enostavna dieta za moderen način življenja je (človek lahko ima samo 1-3 obroke dnevno, napram 5-6 obrokov OH hrane).
Potem pa nekdo reče, da te to lahko prej spravi v grob, po kakšni logiki?
PS, trenutno sicer tečejo študije v smer, ko lahko ima človek normalno OH prehrane in ketone samo dodaja v prehrano in je deležen istih pozitivnih učinkov, kot če bi bil na ketonski dieti, ker s tem zvišuje nivo BHBja v krvi, tkivo pa itak preferira BHB in je potem stanje v telesu skoraj enako, kot če bi bil na ketonski dieti. Amnpak to je šele v povojih, trenutno še ti dodatki niso javno dostopni, je pa zelo blizu temu, mislim da je zadeva že na FDA ali pa bo v kratkem.
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